教師著作

Permanent URI for this collectionhttp://rportal.lib.ntnu.edu.tw/handle/20.500.12235/37073

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    Glycemic and insulinemic responses
    (臺灣營養學會, 2002-01-01) Tang F.C. & Lee
    本研究探討運動前攝食高醣對衰竭運動後血糖與胰島素反應及肌肉恆定之影響。十九位男性大學運動員以相隔一週之時間來進行兩次不同的衰竭運動：正常飲食下進行之衰竭運動是用以測定其最大攝氧量，然後再進行高醣配方飲食（～84%醣類，～13大卡／分斤體重）下之衰竭運動（85%最大攝氧量，10%斜度）；而於運動前30～60分鐘攝食此高醣配方飲食。運動前、中、後採血，運動前、後取尿。高醣飲食後，血漿葡萄糖及胰島素濃度顯著升高（p < 0.0001）。運動中，兩者則顯著下降（p < 0.0001）。運動後，則又逐步回升直到實驗終止；但是，24小時後，血漿葡萄糖濃度則又再次下降到最低點（p < 0.0001）。運動衰竭時，乳酸濃度顯著上升（p < 0.001），衰竭後，則又顯著下降到原點（p < 0.01）。運動衰竭後尿中尿素之排泄顯著減少（p < 0.05），而羥基脯胺酸及3-甲基組胺酸則顯著增加（p < 0.05）。本研究結果認為高度運動前30～60分鐘之高醣飲食並不會導致反應性低血糖症，但高強度運動在能量充足之攝食，仍可能影響到肌肉之恆定性。
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    Influence of branched-chain amino acid supplementation on urinary protein metabolite concentrations after swimming
    (New York, NY : American College of Nutrition, 2006-06-01) Tang, F.C
    The influence of branched-chain amino acid (BCAA) supplementation on urinary urea nitrogen, hydroxyproline (HP), and 3-methylhistidine (3MH) concentrations after 25 min of breast stroke exercise (65-70% maximum heart rate reserved, 65-70% HRRmax) followed by a 600 m crawl stroke competition was investigated in a double-blind, counter-balanced study. METHODS: Male university students (19-22 years old) majoring in physical education participated in the study. Based on the previous swimming time of a 600 m crawl stroke, the participants were divided into two groups: placebo (n = 9, BMI = 24.2 +/- 2.1 kg/m2; 12 g of glucose/day; in capsules) and BCAA (n = 10, BMI = 22.7 +/- 1.5 kg/m2; 12 g of BCAAs/day; in capsules: leucine 54%, isoleucine 19%, valine 27%) groups. The participants maintained a regular dietary intake (except the prescribed breakfast on day 15) and exercise activity at a moderate/low intensity (60-70% HRRmax, swimming and rowing, approximately 1.5 hour/day) during the 15-day study. A prescribed exercise program was performed on day 15. Urinary and blood samples were collected before, during, and after the prescribed exercise for the measurements of the urinary urea nitrogen, HP, and 3MH concentrations in urine, as well as the glucose, lactate, glutamine, alanine, and BCAA concentrations in plasma. RESULTS: Two weeks of dietary supplementation did not induce any changes in the plasma glucose and total BCAA concentrations of either group, nor in the urinary urea nitrogen, HP, and 3MH concentrations in urine. On day 15, after 25 min of breast stroke exercise and a 600 m crawl stroke competition, plasma glucose concentration decreased significantly (p < 0.05) whereas plasma lactate concentration increased significantly (p < 0.05) in both groups. The exercise program prescribed in the study did not affect urinary urea nitrogen, HP, and 3MH concentrations. Twenty hours after the competition, however, a significant increase in the concentrations of urinary urea nitrogen, HP, and 3MH was found in the placebo group (p < 0.05), but not in the BCAA group. CONCLUSIONS: The results obtained in this study suggest that swimming induced muscle proteolysis was prevented by BCAA supplementation. The mechanism could be attributed to the availability of ammonia provided by the oxidation of supplemented BCAAs during exercise.